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10-9-8-7 6 5 4 3 2 1 A shallow and oftentimes maloriented acetabulum causes decreased femoroacetabular contact surface area in DDH. This results in the distribution of shear forces anterosuperiorly in the hip joint onto the acetabular rim. Over time these forces cause degeneration of the acetabular labrum anterosuperiorly and degeneration of the articular cartilage. Here we can see the progression and the development of undiagnosed dysplasia in young age Femoroacetabular impingement is a condition in which extra bone grows along one or both of the bones that form the hip joint giving the bones an irregular shape. Because they do not fit together perfectly, the bones rub against each other during movement. Over time this friction can damage the joint, causing pain and limiting activity. AI is likely a more prevalent underlying cause for the development of hip osteo arthritis. Ganz and colleagues described two different morphological patterns of FAI: cam and pincer. In cam, the predominant morphological abnormality is a thickened, aspherical femoral head–neck junction. When the hip joint is flexed, the cam lesion on the proximal femur abuts against the anterosuperior labrum of the hip, compressing it and pushing it outwards. Meanwhile the acetabular cartilage is compressed and pushed inwards by the shearing force exerted by the cam lesion. The overall effect is separation of the acetabular cartilage from the labrum and delamination of acetabular cartilage from the subchondral bone. In pincer, there is a deepened acetabulum, with acetabular over coverage of the femoral head. As a result the femoral neck abuts against the acetabular labrum, exerting compressive forces that result first in damage of the labrum and eventually the underlying cartilage in a thin circumferential band around the acetabular rim.