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Chronic nonspherocytic haemolytic anaemia is ordinarily not life threatening. It can, however, be disabling and may lead to debilitating complications involving decreased stamina, weakness, cholelithiasis and cholecystitis, iron overload, and progressive hepatic damage. The following case illustrates the clinical picture. In one sense, however, the case is unique. The patient's severe haemolytic disorder (with prominent and persistent signs of increased erythrocyte destruction and production) became the object of a research project leading to the identification of an X-linked inborn error of metabolism arising from a previously unknown form of the key erythrocyte enzyme glucose-6-phosphate dehydrogenase (Figure 11-1).